Healtful eating for weightlifters Essay Example | Topics and Well Written Essays - 1000 words

Healtful eating for weightlifters - Essay Example The awareness for such healthy outlook is more universal now than ever before. There are more fitness camps, more gyms in the neighborhood, more health and fitness equipment and facilities, and those who do not have access to these or do not feel their need have viable alternatives in aerobics and yogic stances. (Robert J. Buresh, MS, et al). The first step towards the achievement of good health and physique is proper diet and exercise. This paper is focused on healthful eating for weightlifters. As such, we are going to concentrate on those muscles that help weightlifters. There are muscles for activities such as marathon runners. These muscles are made up of Type 1 fibres and are ideally suited for runners and other activities of aerobic nature. The Type 2 fibre muscles develop for the purpose of short bursts of energy and are ideal for weight lifters. (Jonny Bowden). Therefore, the diet and exercises for weightlifters are framed after examining two factors. Factor number one is the weightlifter. Diet and exercise comprise factor number two. In order to satisfy the criteria for factor number one, the weightlifter’s age, sex, height, weight, body mass index, present state of health, health history, and disability (if any) are recorded. The recording of data for factor number two will include present diet and physical activities, heart beat and pulse rates, calories level, and health related problems (if any). It may not be possible to obtain the complete regimen of diet and exercise suitable for a particular person in one day. Getting hold of the regimen is the first step. Gradually, the process may undergo changes as the weightlifter continues on the diet and workouts, and upgrades to diets and physical activities which help in performance improvements. The nutrition that results from dietary intake in our bodies is necessary for all of us. The body will simply collapse without nutrition. It is

DISNEYLAND IN QATAR Case Study Example | Topics and Well Written Essays - 3500 words

DISNEYLAND IN QATAR - Case Study Example Market segmentation and target market have been analyzed as part of market analysis. These will form the basis for the development of positioning strategies that are necessary for the appropriate marketing mix to be achieved. The 4ps, which represents Product, Place, Price and Promotion, have been discussed as part of the marketing mix that will be significant for the theme park to establish a unique position in the Qatari market. The strategic fit between the company’s capabilities, resources and the opportunity has been discussed. These are indicators of competitiveness that justify the need for expansion in to the Qatari market. The implementation plan highlights the chronology of events that will lead to the establishment of a fully functional theme park in Qatar. It has been included in the marketing plan as a significant lead to the implementation of proposed activities to accomplish particular objectives over a particular period of time. Each activity corresponds to an objective and output. The implementation plan indicates that establishment and functioning of the theme park will take a period of 14 months. The control strategies that will be employed have been highlighted. These are the actions that the management will be involved in to ensure that the objectives are accomplished according to the implementation plan. Introduction... Disneyland will establish a 500 hectare park with 10 divisions offering different entertainment activities including horse rides, music, live plays, video and wildlife viewing as well as nature trails. This marketing plan aims at maintaining annual revenue of $3 billion. This will be equivalent to 30% of all revenue generated by all five existing Disney’s theme parks in 2011 (White, 2004). Objectives 1. Maintaining 24 hour services during peak season to maintain high sales 2. Increasing the time customers spend in the park through offering half day free for every two days spent at Disneyland during off-peak 3. Promoting institutional visitation by offering special cost reduction during off-peak 4. Encouraging visitation by locals through discounts 5. Establish customer relation management (CRM) to enable the management to keep in touch with clients 6. To increase income by taking advantage of increased number of customers and tourists as a result of Qatar being the host for FI FA Word Cup in 2022 Mission The overall mission of the organization is to maintain leadership among the global providers of entertainment, and more specifically to establish an ultra modern theme park that will outshine any other theme park in the Qatari entertainment industry. The new theme park in Qatar will offer customer focused services to ensure that people get the best value for their money. Market Analysis Market Segmentation Market segmentation has been an important tool for the success of Disneyland theme parks. It involves division of the market in to various segments that consist of groups of consumers with common characteristics. This practice allows the management to know the

Haemolytic Disease of the Fetus and Newborn (HDFN)

Haemolytic Disease of the Fetus and Newborn (HDFN) List and briefly describe three clinical signs for Haemolytic Disease of the Fetus and Newborn (HDFN) Haemolytic disease of the fetus and newborn (HDFN) is a rare disease that occurs when maternal alloantibodies cross the placenta during pregnancy and cause the destruction of fetal red blood cells (RBCs) (Delaney and Matthews, 2015; Haas et al., 2015). HDFN can result in fetal anaemia with progression to severe morbidities, such as ascites, hydrops fetalis, heart failure, kernicterus, and death (Delaney and Matthews, 2015). The clinical presentation of HDFN is variable, in which there are several manifestations that may occur (Murray and Roberts, 2007). Three of the most prevalent clinical signs that allows for neonatal paediatricians to suspect HDFN includes splenomegaly, oedema, and jaundice. HDFN is characterised by the accelerated destruction of RBCs, which results in differing rates of haemolysis and fetal anaemia (Urbaniak and Greiss, 2000). The continuous and rapid process of haemolysis causes extramedullary haematopoiesis, a result of erythropoiesis failing in the bone marrow, in the fetal liver and spleen (Dean, 2005). Organs, such as the liver and spleen, that are involved in the synthesis of RBCs increase the production to combat the rate of destruction and counteract the overall loss (Dean, 2005). The increased workload of the spleen results in its enlargement, termed splenomegaly (Bowman, 1997; Dunn, 1963).          Oedema is an important clinical sign of HDFN that is also associated with the degree of haemolysis and anaemia (Delaney and Matthews, 2015). Oedema can occur due to low levels of serum albumin through a decrease in osmotic pressure (Dean, 2005). Moreover, as the body compensates for fetal anaemia, the fetus can develop a hyperdynamic circulation (Haas et al., 2015). This can result in hydrops fetalis, a severe and life-threatening condition in which there is widespread oedema in the fetal and skin and serous cavities (Haas et al., 2015). A further clinical sign of HDFN is jaundice. Jaunice may occur as a result of haemolysis, in which there is an increase in the level of bilirubin within the body (Urbaniak and Greiss, 2000). Throughout pregnancy, bilirubin is removed via the maternal circulation by the placenta (Dean, 2005). Therefore, a high level of haemolysis may be present with a low level of bilirubin (Murray and Roberts, 2007). However, after birth the haemolytic process continues. At this stage of development, the liver of the neonate is immature and unable to conjugate the excess bilirubin (Urbaniak and Greiss, 2000). The unconjugated bilirubin begins to build and accumulate in the blood of the neonate, causing the skin and whites of the eyes to turn yellow (Dean, 2005). Within 24 to 48 hours after delivery, the level of bilirubin may increase substantially (Urbaniak and Greiss, 2000). Left untreated, this can lead to the development of kernicterus, a condition in which bilirubin deposits form in the basal ga nglia and brain stem nuclei (Haas et al., 2015). Describe the progression of HDFN, from sensitisation to fetal red cell destruction, in a D negative mother carrying a D positive fetus that has not received RhIg. Throughout pregnancy, antibodies from the maternal circulation play a vital role in providing protection for neonates; crossing the placenta from the maternal to the fetal circulation (Dean, 2005). This is essential for the fetus, as by delivery newborns have a relatively immature immune system (Murray and Roberts, 2007). Although the presence of maternal antibodies provides protection, the active transporting of antibodies across the placenta can result in HDFN (Dean, 2005). HDFN is triggered by a sensitisation event. This most often occurs during the first pregnancy; however, it can also arise from a blood transfusion or organ transplant (Delaney and Matthews, 2015). During the course of pregnancy, the maternal and fetal circulations gradually mix with each trimester (Delaney and Matthews, 2015). This results in maternal alloimmunisation, as the maternal circulation has been exposed to foreign RBCs (Haas et al., 2015). Despite the relatively small amount of fetal blood that passes into the maternal circulation, only a small amount is needed for sensitisation to occur (Dean, 2005). This is typical for an RhD negative mother carrying an RhD positive fetus. Sensitisation frequently transpires during the birth of the firstborn RhD positive child, where fetal maternal haemorrhage (FMH) is common (Delaney and Matthews, 2015). However, the risk of sensitisation increases in complicated and extensive labours (Dean, 2005). Sensitisation can also occur through earlier  events in pregnancy, such as a prenatal bleed, trauma, termination of pregnancy, chronic villus sampling, and miscarriage (Sebring and Polesky, 1990). The greatest risk of developing FMH is during the process of labour (Murray and Roberts, 2007). Subsequently, alloantibodies are most likely to form after delivery (Delaney and Matthews, 2015). After sensitisation of an RhD negative mother carrying an RhD positive fetus, the mothers serum will contain anti-D (Dean, 2005). Importantly, the maternal anti-D that is formed is of the IgM class and unable to effectively cross the placenta (Delaney and Matthews, 2015). As a result, HDFN is rare in first-born children and unlikely to have any clinical consequence or significance (Dean, 2005). However, once the maternal circulation has been exposed to the fetal circulation the maternal immune system has the potential to respond to foreign red cell antigens (Delaney and Matthews, 2015). Through extensive research and cohort studies, it has been established that HDFN is most likely to effect subsequent pregnancies (Dean, 2005). Maternal alloantibodies of the IgG1 and IgG class cause significant haemolysis, thus the most clinically significant forms of HDFN (Roberts, 2008). In the event that an RhD negative mother becomes pregnant for a second time, interaction with the RhD antigen stimulates the production of IgG type anti-D, which can be transported across the placenta into the fetal circulation (Delaney and Matthews, 2015). Once anti-D has entered the fetal circulation, it binds to the RhD antigens found on fetal RBCs and labels them to be destroyed (Delaney and Matthews, 2015). From here, the pathophysiology of the disease ensues, as illustrated in figure 4. Explain the action of RhIg in a D negative mother that has a D positive fetal bleed. Rh immunoglobulin (RhIg) is routinely used in clinical practice to prevent HDFN. RhIg is prepared from human plasma that has been immunised to the D antigen and functions by targeting RBCs that are positive for the D antigen (Brinc and Lazarus, 2009). The use of prophylactic anti-D remains the gold standard approach of antibody-mediated immunosuppression, having been used for several decades (Giancarlo et al., 2010). However, the mechanism of action of RhIg is not fully understood and there are three key hypotheses that have been proposed to explain its method of action. Antigen Clearance Hypothesis The first hypothesis is the antigen clearance hypothesis and is considered the main mechanism of action. Here, IgG is understood to prevent an antibody response by increasing the rate of phagocytosis and the removal of RBCs from circulation via the mononuclear phagocytic system, prior to recognition by the immune system (Brinc and Lazarus, 2009). IgG opsonised RBCs are believed to engage in the activation of IgG receptors (FcÃŽÂ ³Rs) on effector cells, stimulating phagocytosis. IgG is also suspected to increase the clearance of RBCs through the stimulation of complement activation on the RBC surface. Anti-D does not activate complement and therefore it is believed that FcÃŽÂ ³R-mediated phagocytosis is the mechanism by which anti-D is cleared (Brinc and Lazarus, 2009). FcÃŽÂ ³RIIB mediated B-cell inhibition hypothesis This mechanism is the most recently proposed and came about through the discovery of increased levels of transforming growth factor-ÃŽÂ ² and prostaglandin E2 in a number of pregnant women who were given RhIG. This mechanism proposes that RBCs and IgG form a complex in which a negative signal is delivered to inactivate antigen-specific B cells. However, mice models deficient in FcÃŽÂ ³RIIBhave shown that the involvement of FcÃŽÂ ³RIIB is not needed to induce antibody-mediated immune suppression. Furthermore, FcR-like molecules have been discovered in both mice and humans. It is believed that the FCRLs mediate the B-cell inhibition, however, this has yet to be demonstrated (Brinc and Lazarus, 2009). Steric Hindrance Hypothesis The Steric hindrance hypothesis proposes IgG binds the antigen, preventing the B-cell receptor from recognising the corresponding epitopes. Most anti-D epitopes are not blocked by RhIg. This allows free D epitopes to be detected after administration of RhIg. Monoclonal anti-D has been shown to prevent antibody responses by binding 10-15% of epitopes. This pathway has not been studied in detail and therefore knowledge of the immunobiology is limited. However, it is believed that IgG binding of D epitopes allows a formation between RBCs and B cells, in which this prevents B-cell activation (Brinc and Lazarus, 2009). The established methodology in the UK for the Quantification of a D positive fetal bleed is via flow cytometry with FITC-anti-D (FITC-BRAD3). Name and describe 3 alternative methods used worldwide to detect fetal bleeds In addition to the use of flow cytometry, several alternative screening methods are available to determine and quantify FMH (Kim and Makar, 2011). Three screening methods that are used worldwide include the rosette screen, Kleihauer-Betke acid elution test, and flow cytometry using anti-fetal haemoglobin antibodies. The rosette test is a screening method that is used to qualitatively detect fetal bleeds equal to or greater than 10 mL and 0.2% of fetal cells present in the maternal circulation (Kim and Makar, 2011). The rosette test works by indirectly identifying the presence of D positive fetal RBCs in D negative mothers (Solomonia et al., 2012). To perform this test, a maternal blood sample is collected, incubated with exogenous anti-D, and washed. D positive RBCs are added and are key as they act as an indicator. The sample is examined using a light microscope. In the presence of fetal D positive cells, the indicator RBCs form aggregates or rosettes around the coated fetal RBCs (Solomonia et al., 2012). A positive result is indicative of an FMH greater than 10 mL and requires quantification by Kleihauer-Betke acid elution test or flow cytometry to determine the dose of RhIg to administer (Kim and Makar, 2011). This test can encounter and produce false-positive results. This largely occurs if the mother of fetus is weak D. Furthermore, in the presence of a direct antiglobulin test (DAT), the rosette test may produce a false-negative result. This can be attributed to crosslinking and agglutination of the mothers antibody coated cells (Kim and Makar, 2011). The Kleihauer-Betke acid elution test is a screening method that differentiates between fetal haemoglobin (HbF) RBCs and adult Hb (Bromilow and Duguid, 1997). The underlying notion of this test is fetal RBCs largely contain HbF and are resistant to acid elution, whereas in contrast, adult Hb is acid-sensitive (Kim and Makar, 2011). To perform this test, a maternal blood sample is taken to prepare a thin peripheral smear. The peripheral smear is dried, immersed in fixative, exposed to and incubated with an acid buffer, and stained with eosin. Under a microscope, the test reveals fetal cells to be stained a dark pink-red colour, whilst adult red cells appear pale or as uncoloured ghost outlines (Kim and Makar, 2011). Under a microscope, the fetal cells are counted and reported as a percentage of adult cells (Kim and Makar, 2011). Flow cytometry using anti-fetal haemoglobin antibodies is a variant of flow cytometry that detects RhD positive fetal cells (Kim and Makar, 2011). In this method, monoclonal antibodies are directed against HbF (Davis, 2007). A maternal blood sample is collected and an RBC count is performed (Davis, 2007). Cells are then fixed and permeabilised with detergent to enable antibodies to enter the cellular membrane and bind HbF (Davis, 2007). A flow cytometer is used to analysed the antibody stained cells (Davis, 2007). This method uses positive and negative controls simultaneously to differentiation between fluorescence from fetal RBCs and non-specific background staining (Kim and Makar, 2011). The positive control is also highly important in setting out the parameters for gating a sample (Kim and Makar, 2011). Rosette test Kleihauer-Betke acid elution test Flow cytometry using anti-fetal haemoglobin antibodies Advantages: Commercial kits Widley available Simple to use Fast Inexpensive Advantages: Not dependent on presence of RhD antigen Requires only basic laboratory equipment Inexpensive Can be used to assess fetal welfare in RhD positive patients Advantages: Quantitative Automated good precisions, sensitivity, accuracy, and reproducibility Cost-effective Less labour intense Disadvantages: Only applicable to RhD negative mothers carrying RhD positive fetus Qualitative only not quantitative Disadvantages: Laborious to perform Accuracy and precision limited due to variation in test characteristics Poor reproducibility Subjective Disadvantages: If mother and fetus have the same RhD type or mother is RhD positive cannot be used to determine FMH False positives due to hereditary persistence of fetal Hb, increased levels of Hb in pregnancy and certain disease states a) A >2mL bleed is detected via acid elution test in a sample taken from a D negative mother. Quantification via anti-D flow cytometry results in a zero bleed. Explain two possible reasons for the opposing results. The differing results of the acid elution test and anti-D flow cytometry may be explained by the RhD status of the mother and fetus. If the mother is not RhD negative and is carrying an RhD positive fetus, this test would not reproduce the results of the acid elution test. Additionally, if the fetus has an RhD negative status, this would also cause the test to fail and detect a zero bleed. Furthermore, flow cytometry cannot accurately detect weak and partial D variants, resulting in a false negative result. Therefore, if the mother or fetus has either of these D variants, FMH would not be detected via flow cytometry. An alternative reason for the differing results between the two tests could be attributed to haemoglobinopathies, in which the flow cytometer detects HbF. Lastly, there could be a mistake in the tube, such that the antibody was not detected, causing an incorrect result. b) Suggest a suitable test alternative to those already conducted to investigate the sample further and explain your reasoning for the alternative test. The use of flow cytometry using anti-fetal haemoglobin antibodies would be a beneficial test to implement in order to clarify the results and detect if a bleed is present. This is important to ensure the appropriate and correct dose of RhIg is administered. Testing the sample using HbF flow cytometry would be useful as flow cytometry using anti-D failed to reproduce the results of the acid elution test. There is a possibility that the acid-elution test produced a false positive result. The acid-elution test is limited in that is has poor accuracy and is prone to variations. The false positive may arise as a result of adult hereditary persistence of HbF, which is known in 1 to 2% of the population. Furthermore, during pregnancy the level of HbF rises by 25%. Therefore, the use of HbF flow cytometry would be able to detect if this is what caused the result. The following bleeds were detected via anti-D flow cytometry in a D negative woman. Using the Mollison calculation work out: The bleed volume in mL The total RhIg dose in each case to the nearest 500 IU The top up RhIg dose required in each case to the nearest 500 IU The Mollison calculation is used to calculate the volume of bleed. To do this, the background of the isotope matched control is subtracted from the number of events obtained in the D positive region, as recommended in the BCSH guidelines. The Mollison equation is as follows: D (+) events D (-) events1800FMH = X -ml X 1.22 Total number of events 1 The equation can be simplified to the following: FMH = % of D positive events x 18 x 1.22 0.81% x 18 x 1.22 = 17.79 mL 18 ml bleed* = 18 x 125 = 2250 IU Rounded to nearest 500 = 2500 IU 2500 IU 500 IU = 2000 IU 0.45 x 18 x 1.22 = 9.88 mL 10 mL bleed = 10 x 125 = 1250 IU Rounded to nearest 500 = 1500 IU 1500 IU 500 IU = 1000 IU 0.091 x 18 x 1.22 = 1.99 mL 2 mL bleed = 2 x 125 = 250 IU Rounded to nearest 500 = 500 IU 500 IU 500 IU = 0 IU This bleed is under 4 mL and therefore no top up is required as 500 IU is routinely administered for a bleed of up to 4 mL. 1.09 x 18 x 1.22 = 23.94 mL 24 mL bleed = 24 x 125 = 3000 IU Rounded to nearest 500 = 3500 IU 3500 IU 500 IU = 3000 IU 0.02 x 18 x 1.2 = 0.43 1 mL bleed = 1 x 125 = 125 IU Rounded to nearest 500 = 500 IU 500 IU 500 IU = 0 IU This bleed is under 4 mL and therefore no top up is required as 500 IU is routinely administered for a bleed of up to 4 mL. References Brinc, D. and Lazarus, A. (2009). Mechanisms of anti-D action in the prevention of hemolytic disease of the fetus and newborn. Hematology, [online] 2009(1), pp.185-191. Available at: http://asheducationbook.hematologylibrary.org/content/2009/1/185.long [Accessed 6 Mar. 2017]. de Haas, M., Thurik, F., Koelewijn, J. and van der Schoot, C. (2015). Haemolytic disease of the fetus and newborn. Vox Sanguinis, [online] 109(2), pp.99-113. Available at: https://www.ncbi.nlm.nih.gov/pubmed/25899660 [Accessed 6 Mar. 2017]. Dean, L. (2005). Blood groups and red cell antigens. 1st ed. [Bethesda, Md.]: NCBI. Delaney, M. and Matthews, D. (2015). Hemolytic disease of the fetus and newborn: managing the mother, fetus, and newborn. Hematology, [online] 2015(1), pp.146-151. Available at: https://www.ncbi.nlm.nih.gov/pubmed/26637714 [Accessed 6 Mar. 2017]. Giancarlo Maria Liumbruno, Angelo DAlessandro, Federica Rea, Vanessa Piccinini, Liviana Catalano, Gabriele Calizzani, Simonetta Pupella, Giuliano Grazzini (2010). Blood Transfus. 2010 Jan; 8(1): 8-16. doi: 10.2450/2009.0108-09 Kim, Y. and Makar, R. (2012). Detection of fetomaternal hemorrhage. American Journal of Hematology, [online] 87(4), pp.417-423. Available at: https://www.ncbi.nlm.nih.gov/pubmed/22231030 [Accessed 6 Mar. 2017]. Murray, N. and Roberts, I. (2007). Haemolytic disease of the newborn. Archives of Disease in Childhood Fetal and Neonatal Edition, [online] 92(2), pp.F83-F88. Available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2675453/ [Accessed 6 Mar. 2017]. Roberts, I. (2008). The changing face of haemolytic disease of the newborn. Early Human Development, [online] 84(8), pp.515-523. Available at: https://www.ncbi.nlm.nih.gov/pubmed/18621490 [Accessed 6 Mar. 2017]. Urbaniak, S. and Greiss, M. (2000). RhD haemolytic disease of the fetus and the newborn. Blood Reviews, [online] 14(1), pp.44-61. Available at: https://www.ncbi.nlm.nih.gov/pubmed/10805260 [Accessed 6 Mar. 2017].      Ã‚  

The Influence of Beck :: Bek David Campbell Musicians Essays

The Influence of Beck One of the most eccentric and talented performed of my time is definitely Beck. I have followed Beck since my young teen years and have found that his music has followed me in every aspect of my life. This soundtrack of my being has become so influential that I look forward to every album as a step in the next direction of my days. Bek David Campbell was born July 8, 1970, in Los Angeles, and came from an exceptionally sturdy music background. His father David Campbell, was a conductor and string arranger giving Beck his strong musical background. His mother, Bibbe Hansen, was an actress who went as far as to work with such artists as Andy Warhol. Also, his grandfather, Al Hansen, was involved with the Fluxus art movement and was best known for launching the career of Yoko Ono. Beck grew up mostly in Los Angeles, also spending some time in Europe and in the Kansas City area with both of his sets of grandparents. A seemingly bad decision to drop out of school in tenth grade led to Beck’s early career as a street performer playing acoustic blues and folk music, as well as trying his hand in the poetry. In 1988, he produced a cassette of home recordings called The Banjo Story, which led to his move to New York in 1989. He soon returned to L.A. to find his calling at rock clubs by playing a few songs in between the regular sets. In 1991, Bong Load Records discovered Beck. The label gave him the idea to mix his eccentric style with hip-hop beats creating the infamous Loser single. This was my first encounter with Beck’s music. I was a young teen that was into the grunge movement by the likes of Kurt Cobain and found the tunes on Mellow Gold, the CD that contain the â€Å"Loser† single, to be amazing. I soon found myself buying a yard sale guitar and teaching myself how to play and mimic Beck’s every move on the folk axe. While I was busy enjoying these sweet sounds, Beck's major-label debut, Mellow Gold, was becoming the â€Å"anthem for the so-called slacker generation†. It quickly climbed into the Top 20 and eventually went platinum. He quickly signed to DGC, the David Geffin Recording Company, and put out two more independent albums Stereopathetic Soul Manure, which consisted of lo-fi noise rock and One Foot in the Grave, material from Beck's 1992 session for K Records. The Influence of Beck :: Bek David Campbell Musicians Essays The Influence of Beck One of the most eccentric and talented performed of my time is definitely Beck. I have followed Beck since my young teen years and have found that his music has followed me in every aspect of my life. This soundtrack of my being has become so influential that I look forward to every album as a step in the next direction of my days. Bek David Campbell was born July 8, 1970, in Los Angeles, and came from an exceptionally sturdy music background. His father David Campbell, was a conductor and string arranger giving Beck his strong musical background. His mother, Bibbe Hansen, was an actress who went as far as to work with such artists as Andy Warhol. Also, his grandfather, Al Hansen, was involved with the Fluxus art movement and was best known for launching the career of Yoko Ono. Beck grew up mostly in Los Angeles, also spending some time in Europe and in the Kansas City area with both of his sets of grandparents. A seemingly bad decision to drop out of school in tenth grade led to Beck’s early career as a street performer playing acoustic blues and folk music, as well as trying his hand in the poetry. In 1988, he produced a cassette of home recordings called The Banjo Story, which led to his move to New York in 1989. He soon returned to L.A. to find his calling at rock clubs by playing a few songs in between the regular sets. In 1991, Bong Load Records discovered Beck. The label gave him the idea to mix his eccentric style with hip-hop beats creating the infamous Loser single. This was my first encounter with Beck’s music. I was a young teen that was into the grunge movement by the likes of Kurt Cobain and found the tunes on Mellow Gold, the CD that contain the â€Å"Loser† single, to be amazing. I soon found myself buying a yard sale guitar and teaching myself how to play and mimic Beck’s every move on the folk axe. While I was busy enjoying these sweet sounds, Beck's major-label debut, Mellow Gold, was becoming the â€Å"anthem for the so-called slacker generation†. It quickly climbed into the Top 20 and eventually went platinum. He quickly signed to DGC, the David Geffin Recording Company, and put out two more independent albums Stereopathetic Soul Manure, which consisted of lo-fi noise rock and One Foot in the Grave, material from Beck's 1992 session for K Records.

Operating Manual for Spaceship Earth

This short essay contains an outline of the pertinent points and concepts discussed by R. Businessmen Fuller In his essay Operating Manual for Spaceship Earth, which puts forward one argument for evolution. ‘Earth is only eight thousand miles in diameter, which is almost a negligible dimension in the great vastness of space. Our nearest star – the sun – is ninety-two million miles away. The nearest star after that ‘is one hundred thousand times further. It takes approximately four and one-third years for light to get to us' (Fuller: 1969: Pl) It puts us Into perspective.We are Just one of nine planets In one of one Right now, we are traveling at sixty-thousand miles an hour around the sun, as well as spinning axially, which adds approximately one thousand miles per hour to our motion. Each minute we both spin at one hundred miles and zip in orbit at one thousand miles. ‘ However our earth is so ‘extraordinarily well invented' that we don't feel t his. To our knowledge, humans have been on earth for two million years' and not even been aware of this. ‘Its so superbly designed' that It Is able to keep life regenerating despite' the fact that it should be impossible.Fuller: 1969: Pl ) To our knowledge, the sun has to be at Just the right distance away from us to be able to emit the energy we and our plants need to survive but not too close as to fry us. We also have to have the perfect amount of gravitational pull to hold us to the earth. Too much and we wouldn't be able to move but not enough and we would float off Into space. If either one of these were to be slightly out of place we wouldn't exist. This, from our knowledge to date 18. 10. 13, is proven by the fact that in our Milky-way we are the only planet with life form.That's not to disprove that in the future we loud find other life forms existing on other planets in other galaxies. It is statistically probable that there are other planets homing other species bec ause of the fact that there are approximately one hundred billion other galaxies. There are approximately 8. 7 million different animal species on earth (Moral, C et all 2011) plus another 400,000 different species of plant Offers: 2008) All of these survive on the earths surface. However there's not just one of each species.For example, there are approximately 7,186,560,610 humans on this planet (accurate at 10:ma'am 18/10/13). Current World Population: 2013) If we assume that every species has that number of participants then you are looking at 6. 5397702 x ION 6 (calculated by humans x animal species + humans x plant species) That's a lot for one planet to keep alive. Its amazing that we even exist and yet we do. Not only do we exist but we have evolved. ‘Quite clearly, all of life as designed and born is utterly helpless at the moment of birth' and humans ‘stay helpless for a lot longer' than any other species. ‘intellect. We had to experience and learn how to contend with difficulties and wrought ‘comprehensive reviews of the compound facts of experiences using intellect' we found ‘awareness. ‘ We can differentiate between the red berries that will kill us and the red berries that will nourish us. ‘ (Fuller: 1969: UP) We have also learnt to cultivate more of these botanical edibles by genetically inbreeding (Fuller: 1969: Pl) ‘Only as [man] learned to generalize fundamental principles of physical universe did man learn to us his intellect effectively. ‘ (Fuller: 1969: UP) We have been able to discover and understand science. That we are endowed with such intuitive and intellectual capabilities' which we have gained from experiences and the earth have meant that we are able to discover fundamental principles governing the fundamental design of life. ‘ (Fuller: 1969: UP) For example, R. N. A (Ribonucleic Acid) and D. N. A (Deoxyribonucleic Acid) and in discovering these we have been able to ‘ increase our survival potentials millions fold. ‘ (Fuller: 1969: UP) In conclusion, we are only one small species on one tiny planet in the ‘great vastness of space' and so really we are inconsequential.Yet what we have achieved, the percussions and effects we create Just go to show that whilst we may be small we are powerful. In the past 2,000 years we have evolved from thinking the earth is flat to realizing that not only is the earth round but that it is part of a universe. From this we have created rockets that can take us out of our earths atmosphere and into space. Give us another 500+ years and we will have achieved Warp Speed technology. The fact that our planet can have, sustain and regenerate millions of life forms only emphasizes our potential and proves how much more we can learn.

Presentation: Barack Obama “Dreams from My Father”

Barack Obama – â€Å"Dreams From My Father-A Story of Race and Heritance† Today I want to introduce you United States President Barack Obama’s first memoir, dreams from my father. It was first published in July 1995 as he was preparing to launch his political career and republished in August 2004. Author: Since my book is an autobiography I won’t tell you everything about the author, I just will give some personal facts about him. Barack Obama was born in 1961 in Honolulu, Hawaii to Barack Obama Senior and Ann Dunham. In 1991, Obama graduated from Harvard Law School.While in law school he worked as a co-worker at the law firms of Sidley & Austin where he met his wife, Michelle, whom he married in 1992. Barack and Michelle Obama have two daughters: Malia Ann and Natasha, known as Sasha. On November 4 2008, Obama won the presidency of the United States. The inauguration of Barack Obama as the 44th President took place on January 20, 2009. On October 8 of las t year, Obama was named the 2009 Nobel Peace Prize laureate. Characters: Paternal Family The Obamas are members of the Luo, Kenya's third-largest ethnic group. Hussein Onyango Obama Barack Obama's paternal grandfather †¢Habiba Akumu Obama Barack Obama's paternal grandmother and the second wife of Hussein Onyango Obama †¢Barack Hussein Obama Senior He is the father of Barack Obama. Visited his son only for one single time. In his memoir, Barack calls him the Old Man. †¢ Kezia †¢Jane Her sister, Jane, is the ‘Auntie Jane' mentioned at the very start of Dreams from My Father when she telephoned President Obama to inform him that his father had been killed in a car accident. †¢Ruth NdesandjoBorn Ruth Nidesand, in US, Barack Obama Sr. ‘s third wife Maternal Family †¢Ann Dunham Mother of Barack Obama. His mother is open minded, idealistic, naive in one sense, world-wise in another. †¢Stanley Dunham is the grandfather of Barack Obama. Stanley and his wife Madelyn raised Obama in Honolulu, Hawaii. His grandparents love him unconditionally. He learns his grandfather's strengths and weaknesses, but never really comes to see him as the father figure he's seeking. †¢Madelyn Lee Payne Dunham Barack Obama's maternal grandmother. †¢Lolo SoetoroStepfather of Barack Obama, born in Indonesia, Obama speaks fondly of his step-father, and learns several life lessons from him, but unfortunately his mother's relationship with his step-father doesn't last. Plot: 1. Origins 2. Chicago 3. Kenya Epilogue Barack Obama, the current narrates a heart-rending story about his personal implications; tribulations in the American society, and his father’s roots. Ad 1. The story opens up in New York, where he hears that his father—a figure he knows more as a myth than as a man—has died in a car accident.Two years later after Baby Obama was born, his father won a scholarship to continue with studies at Harvard University . And so, Obama Senior left Hawaii, went to Harvard, separated from his small family and, was divorced by Ann. He completed his studies and returned to Kenya to take up a senior job at the department of national planning. Baby Obama never saw him until much later. As a young boy, his mother marries a man from Indonesia and they go to live there. His mother sends him back to Hawaii to live with his grandparents so he can attend a prestigious Hawaiian school, as one of only three black students there.Obama has one encounter with his father when he's ten in Hawaii. Obama is a little adored, a little overwhelmed by his father’s visit. Nevertheless his father doesn’t stay very long and leaves Obama with more questions than answers. Ad 2. Propelled by a desire to understand both the forces that shaped him and his father’s legacy, the book then moves on to moves to Chicago where he works as a community organizer. It's challenging work that is rarely rewarding, but Obam a gives it his all. Then a relative from Kenya, his aunt Jane, calls to tell him his father has died, but Obama's not quite sure how to feel about that or how to react.Several weeks later, his Kenyan half-sister, Auma, makes contact with him. Auma gives him a sneak peek into his father's life in Kenya. Obama is fascinated by the life Auma leads and wants to learn more about their father. Before he leaves community organizing to go to Harvard Law School, he makes arrangements to visit Auma in Kenya and TO TRACE THE ROOTS OF HIS FATHER. Ad 3. In Kenya, Obama discovers a family he didn't even know. His father had at least four wives, and Obama has a slew of brothers and sisters who are living in their father's shadow.Obama and Auma visit with one their grandfather's wives, â€Å"Granny. † She tells Obama's father story to him. The story: Because Onyango wanted his son to be provided as best as possible as for opening up a good future, his father received a scholarship in economi cs through a special program which offered Western educational opportunities to outstanding Kenyan students. Following Obama Sr. enrolled at the University of Hawaii where Obama Sr. married Ann Dunham, though she would not find out that her new husband was already married to a pregnant wife until much later.Ann quit her studies to care for the baby, while Obama Sr. completed his degree, leaving shortly thereafter to study at Harvard University. While studying at Harvard, Obama Sr. met an American-born teacher named Ruth who he married after divorcing from Barack’s mother. After a while Obama Sr. published a paper entitled â€Å"Problems Facing Our Socialism† harshly criticizing the concept for national planning. This conflict with President Kenyatta destroyed his career. Following he was fired from his job, was blacklisted in Kenya, and began to drink.He had a serious car accident, spent almost a year in the hospital, and by the time he visited his son in Hawaii, when Barack was ten years old. Obama Sr. ‘s life fell into drinking and poverty, from which he never recovered. Obama Sr. later lost both legs in another automobile collision, and subsequently lost his job. He died at the age of 46, in a third car crash in Nairobi. From then on, Obama realized that the man he thought to be a failure was in fact a hero in his village. His father was the first person to have gone to the US University from his village.Then, Obama forgave his father and decided to change his life basing on his father’s dreams. Themes: †¢Racism Throughout the whole book the main character is faced with Racism. From early childhood he had to struggle with students that teased him when he was playing with one of the three black kids. Even in Kenya, when he and some of his family members are at a restaurant, the waitress who is black, ignores their orders and doesn’t serve them after she has seen European tourists who are white. †¢Search for communi ty As a young adult, Obama set off in search of community and purpose, with the reat role models of the civil rights movement. The glory days of the civil rights movement were long gone when Obama gets an organizing job in a poor neighborhood on Chicago's South Side plagued by crumbling public housing, disappearing manufacturing jobs, and rising crime. The group's founder is a Jewish man who is not fully trusted by the community. At the same time Obama makes personal connections. He becomes close with the three middle-aged African-American women who are core to the organization, and develops a friendship with an eccentric, pot-smoking Catholic organizer.He looks out for Kyle, the teenage son of a volunteer who is in danger of getting into trouble. One of the most moving bits in the book where Obama tells the group he is headed off to Harvard Law School, and promises his friends in the neighborhood that he'll be back. †¢Search for family and identity Feeling out of place in high school, Obama gravitates toward the black kids and works to embrace an African-American culture that matches others' expectations of his appearance, but is different from his upbringing and background. A trip to Kenya before law school is an opportunity for discovery.Obama grew up with an idealized vision of his father, which both intimidated and inspired him. As he gets to know his African family, he finds out that his father's life was more complex and less perfect than the idealized image. In Kenya, Barack Junior finds a family that is loving, close, and welcoming but surrounded by problems — feuds, alcoholism, and poverty. The stories that Obama hears on his trip make things more complicated, not simpler. The stories provide context for the personality flaws, passions, that which are more meaningful, more admirable, and more forgivable, than a shallow but false idealized image. The Limits of â€Å"Organizing† After a series of infamous defeats, the persistence, sk ill and empathy of Obama's group begins to pay off. They organize cleanup for the housing project, job training for the neighborhood, mentoring for school kids. But in the end, during a public forum where the neighborhood people demand basic maintenance for public housing projects, the bureaucrats explain that the Housing Authority budget allows a asbestos removal, or basic repairs, but not both. So as you can see, one small person can’t change everything without reaching some limits.